And some doctors are claiming that Pristiq should simply be “forgotten about“. [push]Quite frankly, Effexor has a lot of side effects and withdrawal issues associated with it, so it wouldn’t surprise me if even a slightly cleaner version would be better tolerated.[/push] And most importantly, if you and your mental illness fall into the category of people who are helped by Pristiq, who were not helped with Effexor, you really . Let doctors be concerned about drug politics on their own time. Those of us who have been in treatment for a mental illness for a while know that it’s all a crap shoot anyway, so why take away an option that might get you better? Note #1: All that being said, if you’re successful on Effexor, don’t just switch to Pristiq, those other chemicals in Effexor might be the ones helping you. It has absolutely brutal withdrawal symptoms that (for me) kick in within two hours of expected dosing time.And that group of people is precisely why doctors should forget about Pristiq, or any other drug. No one can predict whether you’ll have the same reaction to Pristiq. I gather this also happens with Effexor, and Effexor’s withdrawal effects tend to be even worse.
Achieving remission is critical because clients who do improve but continue to experience residual symptoms of depression Antidepressants should be used prudently in the management of depressive symptoms associated with bipolar disorder, so clients who do not improve on antidepressants or even get worse would likely benefit from a diagnostic re-assessment for bipolar.
So, reduced uptake causes prolonged effect of serotonin and norepinephrine in the brain.
Unlike antidepressants of the past, Pristiq is approximately ten times more potent at inhibiting serotonin uptake than norepinephrine uptake.
In other words, it’s an antidepressant that works on both serotonin and norepinephrine neurotransmitters.
It is not the only antidepressant to do this, but SNRIs are a smaller class of drugs than those that just effect serotonin alone (like Prozac).